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Genetic markers in GIPR and GLP1R pathways may provide context, though they do not predict exact medication response and should inform, not replace, clinical judgment

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Among the SK channel members (SK1, SK2 and SK3), SK3 is expressed predominantly on neutrophils and the knockdown of SK3 in differentiated HL-60 (dHL-60) cells reduces the NETs formation induced by ionomycin-mediated NOX-independent NETosis

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