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Suppression of the murine osteoblasts MC3T3-E1 proliferation by dexamethasone (Dex) was associated with G1 phase delay and apoptosis induction accompanied by p53-dependent activation of p21 and the up-regulation of proapoptotic genes NOXA and PUMA [24]

36,37 As a result of the activation of stellate cells and a subsequent enhancement in collagen and extracellular matrix synthesis, the space of Disse becomes thickened, so that capillarization develops and the normal fenestrated architecture of the sinusoidal endothelium is lost