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A small molecule inhibitor of nicotinamide N-methyltransferase for the treatment of metabolic disorders

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Research has focused on amylin receptor agonism (AMY1, AMY2, AMY3) and downstream effects on food intake, gastric emptying and energy homeostasis

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Although TET members such as TET2 functions as tumor suppressor in certain cancers including AML and glioma 45 , we showed that knockdown of individual TET members consistently abrogates the YBX1AMPK axis in liver and lung cancer cells, suggesting potential functional synergy among TET members in regulating local DNA demethylation, although the underlying mechanisms warrant further investigation