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Insulin resistance as a link between amyloid-beta and Tau pathologies in Alzheimers disease

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Its mechanism of action involves simulating the effects of GLP 1, an endogenous hormone that prompts insulin release and reduces glucagon secretion from the liver2, by activating the GLP 1 receptor

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ROS generation occurs via multiple metabolic pathways that are tightly controlled by oxidant and antioxidant signaling and plays an important role in modulating inflammation during viral infections 21,22