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In this review, we aim to explore the emerging connections between ApoE genotype and pathogenic proteins in individuals with AD and other neurodegenerative disorders

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Studies indicate it interacts with blood vessels, nerves, and connective tissue in experimental models

extend as needed for ongoing management

Epigenetics of dementia: understanding the disease as a transformation rather than a state

Once the combined oxidative capacity of this system is exceeded, LD expansion becomes insufficiently protective and may convert into a feed-forward mechanism sustaining NEFA accumulation, ROS generation, bioenergetic collapse, and necroinflammation ( 3.6 Mitochondria and secretory granules Because pancreatic injury is ultimately expressed through disturbed secretion and premature enzyme activation, the consequences of mitochondrial dysfunction must also be considered within the apical secretory domain