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256 The pathogenic mechanisms by which gluten gliadin peptides induce inflammation in remote organs, and the extended potential harmful effects in non-celiac ADs are described in Figure 1 Fig

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Specific genetic variants in the GLP-1 receptor gene (GLP1R), the glucose-dependent insulinotropic polypeptide receptor gene (GIPR), and metabolic regulators like FTO influence appetite suppression, insulin sensitivity, and satiety signaling

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