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Moyer 2011)
While both GSH and GABA have been implicated in various neurological disorders, the current study provides valuable insights into the specific context of ASD and highlights the need for further research to explore other neurochemical alterations that may contribute to the pathophysiology of this complex disorder
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Knowledge gaps include the polyamine pathway synthesis of GABA and the specific calcium channels activated by GABA, with limited exploration of GABA's signaling role through ROS (Guo et al
Neuroprotection and Brain Health GHK-Cu shows neuroprotective potential by crossing the blood-brain barrier and promoting neuronal growth and repair
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