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[19] [20] UK practice generally supports continuing GLP-1 receptor agonists perioperatively with appropriate risk assessment and aspiration precautions, rather than routine discontinuation

Diabetes increases expression of pro-apoptotic Bax and phosphorylation of JNK, while suppressing anti-apoptotic Bcl-2, thus promoting cell death of germ cells, Sertoli cells, and Leydig cells [241]

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siRNA-mediated knockdown (KD) of EPAC2 alone resulted in inhibition of growth in these transformed melanocytes, similar to the combined treatment of siEPAC1/2 (Fig