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Recent studies have explored the potential of GLP-1 receptor agonists (GLP-1 RAs) as therapeutic agents for managing T2DM and its associated cognitive deficits [9,10]

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Shifting trends in the indication of glucagon-like peptide-1 receptor agonist prescriptions: a nationwide analysis

This is supported by cell-killing assays which indicate increased lysis of NK cell-sensitive lines after GLP-1 treatment, alongside heightened production of IFN- and granzyme B [173]

It is well documented that GSTs conjugate GSH to an array of electrophilic compounds of exogenous and endogenous origins (Perperopoulou et al., 2017)