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Today, GLP-1 drugs improve blood sugar control and promote weight loss, transforming metabolic disease management

We show that the cardioprotective actions of GLP-1(2836) do not depend on a functional transmembrane GLP-1R but rather are mediated intracellularly through type 10 soluble adenylyl cyclase (sAC), accompanied by increased cAMP levels, protein kinase A (PKA) activation, and endothelial nitric oxide synthase (eNOS) phosphorylation

S-Acetyl-Glutathione is the first stable form of glutathione, meaning it resists stomach acid and thus reaches the cells intact

Partly through these actions, GLP-1 receptor agonism was shown to reduce cardiovascular disease (CVD) in people with T2D with previous CVD or at high-risk thereof

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