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Multiple studies show BPC-157 accelerates healing of gastric and duodenal ulcers through several mechanisms: increasing blood flow to damaged areas, reducing acid secretion, enhancing mucus production, and promoting epithelial cell migration

Probiotic-derived EPS activate TLR2 on DCs, fostering a tolerogenic phenotype characterized by IL-10 secretion and reduced autoreactive B/T cell activation (108, 146)

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Measures of antioxidant status of the horse in response to selenium depletion and repletion

57.1%, p = 0.10)